Plamsa 5=-AMP (pAMP) and uric acid (UA) elevated in type 2 diabetic
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Frontiers The Na+/K+-ATPase: A potential therapeutic target in cardiometabolic diseases
Identification of pathognomonic purine synthesis biomarkers by metabolomic profiling of adolescents with obesity and type 2 diabetes
Sodium-Glucose Cotransporter-2 Inhibitors in Reducing Uric A
08 Diabetes by Urvi Trivedi - Issuu
Purine metabolism. Adenosine monophosphate (AMP) is converted to
AMP-activated protein kinase α2 contributes to acute and chronic hyperuricemic nephropathy via renal urate deposition in a mouse model, European Journal of Medical Research
AMPK alleviates high uric acid-induced Na+-K+-ATPase signaling impairment and cell injury in renal tubules
AMP-activated protein kinase α2 contributes to acute and chronic hyperuricemic nephropathy via renal urate deposition in a mouse model, European Journal of Medical Research
Cells, Free Full-Text
Gut bacteria impact host uric acid burden and its association with atherosclerosis
PDF) HbA1c does not reflect prandial plasma glucose excursions in type 2 diabetes
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